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doi: 10.1016/j.jplph.2014.12.014 4 ChenI

This is due to the elevated cellular vulnerability to oxidative stress and reactive oxygen species ( Specifically in regard to T lymphocytes, GSH is necessary for T cell phenotype and effector functions

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Excessive generation of ROS within ovarian, adipose, and hepatic tissues overpowers antioxidant defenses, leading to oxidative damage of lipids, proteins, and mitochondrial DNA (74)

Altered mitochondrial fusion drives defensive glutathione synthesis in cells able to switch to glycolytic ATP production